Farmworkers are dying from extreme heat

Grist – This story was originally published by Mother Jones and is reproduced here as part of the Climate Desk collaboration. Also: https://academic.oup.com/ndt/article/32/4/598/3604969

On June 16, Miguel Angel Guzman Chavez arrived in Georgia from Mexico. He was 24 years old and went right to work picking tomatoes. The Georgia heat was consistently more than 90 degrees, and on June 21, the temperature soared to 95 degrees. That day, Chavez collapsed in the field, suffering from heat stroke, which then led to cardiac arrest. Less than two hours later, he was pronounced dead at the Colquitt Regional Medical Center.

“People don’t realize what a serious problem this is,” David Arkush, managing director of the climate program at Public Citizen, tells Mother Jones. When farmworkers are out picking tomatoes or spraying pesticides in the high heat, they can be exposed to heat-related illnesses, some of which can lead to death if left untreated. According to Public Citizen, 130 million workers who make their living outside — from farmworkers to construction workers — lack heat stress protections. Between 1992 and 2016, nearly 70,000 workers were seriously injured from heat, and 783 of them died.

Since farmworkers are often from marginalized groups like undocumented immigrants — the Department of Labor estimates 47 percent of them are undocumented — fear of deportation can lead to a reluctance to report incidents or even seek medical attention. That means the real numbers may be even higher. Amid the Trump administration’s crackdown on immigrants coupled with the reform of the visa system, many farmers have been struggling to find workers.

The overwhelming majority of scientists agree that human-made climate change is making the planet hotter. According to NASA and National Oceanic and Atmospheric Administration data complied by Climate Central, a nonprofit news organization that analyzes climate data, the 10 warmest years on record have all occurred since 1998. More alarmingly, the five hottest years have all been in the 2010s, with 2016 being the warmest year ever recorded. With global warming come extreme weather conditions, including unprecedented heatwaves that can turn deadly. Every demographic will feel the effects of rising temperatures, but people who work outdoors — like Chavez and the estimated 2.5 million farmworkers in the United States — are already feeling the effects.

Those exposed to high heat are susceptible to a variety of heat-related illnesses ranging from rashes and cramps to heat stroke that damages the kidneys and brain. If steps aren’t taken to cool the body down immediately, extreme heat can also lead to death. The long-term effects have not yet been firmly established, but there is emerging evidence that working outdoors in extreme temperatures can also lead to chronic kidney disease.

The federal government requires each employer to maintain a safe workplace, but it offers very few specific protections for outdoor workers at risk of heat illnesses. On the state level, only three states have issued guidelines for employers on ways to protect their workers from heat stress, but those guidelines lack any power of enforcement. Together with United Farmworkers and Farmworker Justice, Public Citizen has launched a campaign to petition the Occupational Safety and Health Administration (OSHA) for uniform national standards that can protect workers from heat-related illnesses and injuries.

In 2005, the California legislature became the first in the country to pass standards for protecting workers against heat stress. These regulations require employers to provide access to shade and drinking water and to enforce mandatory periods of rest during work shifts. In 2012, the United Farm Workers of America sued the California Division of Occupational Safety and Health, alleging that between 2005 and 2012, 28 farmworkers died from heat-related illnesses. “In farm working, there’s a huge number of migrant laborers, immigrants, and people who don’t speak English,” Arkush explains. “And companies feel like they can get away with exploiting those people.”

Heat-related injuries and deaths among farmworkers cast light on the terrible vulnerabilities these workers face. In 2008, California had six heat-related deaths in the field. Maria de Jesus Bautista, a 62-year-old farmworker in Thermal, California, was picking grapes in 110-degree weather. She returned home that evening with a headache and high fever. Her son, Francisco Alvarez Bautista, found her wrapped in a blanket, lying on the sofa, complaining about being cold — despite the soaring temperatures that day. Maria died two weeks later.

After her death, her son said she felt pressured to keep pace with her coworkers. “Some of the ways in which workers are at risk is just the pace of work,” explains Virginia Ruiz, director of occupational and environmental health at Farmworker Justice. “When workers are paid by the amount they pick, there’s a reluctance to take a break.”

In 2013, California state officials shut down the Etchegaray Farms after a farmworker collapsed and died on a citrus orchard outside of Bakersfield, California. Juan Ochoa was checking irrigation equipment in 106-degree weather when he collapsed. Ochoa, who was 37, was from Mexico and had been working for almost a year. When officials came to inspect the farm, they found several violations of California’s heat regulations, including no access to shade. The farm was allowed to reopen once it was compliant.

These tragedies could likely have been avoided if the workers had been required to take regular breaks in the shade — which can often be the difference between life and death. The Centers for Disease Control and Prevention recommends rest periods for workers to hydrate and longer rest breaks for newer workers or those who haven’t grown acclimated to being outside for long periods of time. That, Ruiz says, is where you see most of the major injuries and fatalities.

In the petition to OSHA, the groups are asking for fairly simple protections to limit deadly exposure to the heat. “The most important protections to provide are to require access to rest, shade, and water breaks,” Arkush says. Knowing what to look for and how to work in the heat are also important aspects of working in agriculture that could help save lives, because, Arkush notes, workers often receive no “training or information about warning signs [or] how to prevent stress.” California requires sharing this information, as well as mandatory rest and water breaks, but there are often violations of the law. “There’s noncompliance,” Ruiz says, “and in isolated and rural areas, there aren’t enough inspectors going out there.” In 2016, California’s division of occupational safety and health reported only 62 percent of agriculture employers were complying with heat regulations.

With rising temperatures, the problem will only get worse if the federal government doesn’t step in. “It’s entirely preventable,” Ruiz says of heat-related illnesses, “but I think we’re going to see them more frequently as we have these spikes in temperature across the country.”

This story was originally published by Grist with the headline Farmworkers are dying from extreme heat on Aug 24, 2018.

Pro: Heat stress as a potential etiology of Mesoamerican and Sri Lankan nephropathy: a late night consult with Sherlock Holmes 

Nephrology Dialysis Transplantation, Volume 32, Issue 4, 1 April 2017, Pages 598–602,https://doi.org/10.1093/ndt/gfx034
Published:
12 April 2017

ABSTRACT

Epidemics of chronic kidney disease are now recognized in Central America, Mexico, India and Sri Lanka, and there is also some evidence that similar epidemics may be occurring in the USA, Thailand and elsewhere. A common denominator for each location is manually working outside in extremely hot environments. Here we review the evidence that the primary etiology may be heat stress related to repeated subclinical or clinical acute kidney injury that eventually manifests as chronic kidney disease. In some aspects, the disease may manifest as subclinical heat stroke, subclinical rhabdomyolysis or a subclinical tumor lysis syndrome. While toxins could be involved, it would be difficult to attribute this as a main mechanism, given the wide range of occupations and geographic regions manifesting this disease. While some of the epidemics may be due to better reporting, we believe the most important reasons are increasing heat extremes (heat waves) coupled with hydration with sugary or, less commonly, alcoholic beverages.

In my dream last night, I once again had the opportunity to visit 221B Baker Street to consult with my friends, the honorable Dr John Watson and Mr Sherlock Holmes. Dr Watson met me at the door, wearing his Oxford tweed jacket with bowtie, this time appearing a little bit more portly than before.

‘Good to see you, Johnson. Here to talk about sugar again?’ Watson said with a smile as he offered a tray of chocolates to me.

‘No, Watson, not this time’, I said as I quickly took a chocolate. ‘But I have been asked to defend an argument, and I could use some advice.’

Watson took me back to the living room den where I found Holmes in his usual chair, reading a book on maritime history as he smoked his pipe. As I entered the room, Holmes looked up and cracked an almost imperceptible smile.

‘Holmes, there is an epidemic of chronic kidney disease (CKD) that has emerged since the 1970s in Central America. It tends to affect sugarcane workers living on the Pacific Coast. Similar epidemics are occurring in India and Sri Lanka, and there is some evidence that it is more widespread and affects other areas in Southeast Asia, sub-Saharan Africa, Mexico and certain regions in the USA. So far more than 40 000 people have died [12].’

‘It sounds like it could be a toxin. There are so many chemicals used these days, the wells are likely contaminated. Has testing been done?’ Watson spoke.

‘Yes, that is the hypothesis I am debating against—it certainly is an emotional issue, as the workers are subjected to harsh working conditions and have lots of exposures. Yet to date, no toxin has been identified, and while there are drugs, toxins and infections that may cause CKD, the clinical characteristics do not fully mirror the current epidemics’ (Table 1).

Table 1

Toxins and infections associated with acute and CKD

Etiology Past epidemic Epidemiology Acute Chronic Biopsy Ref
Lead Queensland (1920s) Children (paint)
  • Neuro (wrist drop, ocular palsies)
  • Abdominal colic
  • Ocular (optic neuritis)
  • Fanconi syndrome
  • CKD
  • Hypertension
  • Gout
  • Anemia
  • CIN;
  • Glomerulosclerosis Microvascular lesions
[3]
Cadmium Toyama (1930–60s)
  • Contaminated river
  • Rice paddies
  • Bone pain
  • Glycosuria, proteinuria Hypophosphatemia
  • Osteomalacia
  • Fanconi, glycosuria
CIN [4]
Arsenic Manchester (1900s) Beer contamination
  • Abdominal pain
  • Neural and vascular
  • Hyperkeratoses
  • ?Albuminuria, CKD
[56]
Leptospirosis Philippines (2009) Water contamination
  • Conjunctival suffusion
  • Myalgias and fever
  • Acute interstitial nephritis
  • Jaundice, meningitis (rare)
?CKD [78]
Hantavirus Korean War (1953) Field mouse
  • Fever, myalgias, petechiae
  • Albuminuria
? CKD AIN, hemorrhage [9]
Aristolochia Balkans (1940–2000), Brussels (1990)
  • Contaminated wheat
  • Contaminated ‘slimming herbs’
  • Proteinuria, glycosuria
  • Rapidly rising creatinine
  • CIN, uroepithelial CA;
  • Mild hypertension
  • Anemia
CIN [10]
Ochratoxin A ?Northern Africa
  • Contaminated foods
  • Cereal, nuts
Glycosuria, proteinuria CIN? CIN (animals) [11]
Analgesic abuse Australia, Belgium (1950–80s) Phenacetin-containing analgesic combinations
  • AIN, papillary necrosis, sterile pyuria, hematuria
  • Polyuria
CIN, hypertension, uroepithelial CA CIN [12]
Organophosphates (anticholinesterases) Agrochemicals
  • Salivation, diarrhea Abdominal pain Tachycardia, tremor
  • Seizures, AKI (rare)
?CKD ?
Paraquat Glyphosate
  • Herbicide
  • Agrochemical
  • ARDS, rare AKI, liver
  • Abdominal pain, AKI, liver failure, low BP
  • ?CKD, Parkinson’s
  • ?CKD
[1314]

Key: AKI, acute kidney injury; ARDS, acute respiratory distress syndrome; CKD, chronic kidney disease; CA, cancer, CIN, chronic interstitial nephritis.

‘Emotional qualities are antagonistic to clear reasoning [The Sign of the Four]’, said Holmes sternly to Watson as he blew a smoke ring into the air. ‘As a rule, the more bizarre a thing, the less mysterious it proves to be [The Red-headed League]. I make a point of never having any prejudices, and of following docilely wherever fact may lead me [The Reigate Puzzle]. So always approach a case with an absolutely blank mind. It is always an advantage. Form no theories, just simply observe and draw inferences from your observations [The Adventure of the Cardboard Box].’

‘Holmes, we do have some observations that have led to the hypothesis that heat stress may be the cause. I realize that it is not necessarily a sexy cause, but the data appear strong’, I said quietly.

‘Do not bias my mind by suggesting theories or suspicions. I wish you simply to report facts in the fullest possible manner to me, and you can leave me to do the theorizing [The Hound of the Baskervilles]. Before we start to investigate, let us try to realize what we do know, so as to make the most of it and to separate the essential from the accidental [The Adventure of the Priory School]. [The best approach is to reason backward from effects to causes [The Adventure of the Cardboard Box]’. [So give me] Data! Data! Data!’, Holmes cried impatiently. ‘I can’t make bricks without clay [The Adventure of the Speckled Band].’

‘So, a common finding is that the disease is occurring in areas of the world that are extremely hot, exposing the workers to temperatures that are considered dangerous for prolonged work. Indeed, heat exposure is always the common denominator, for while the disease is highest in sugarcane workers, in the epidemic in Central America it has also been observed in construction workers, port workers, fisherman and those working in other agricultural occupations (banana, cotton). There is also the finding that sugarcane workers who are working at higher altitudes where the temperature is cooler have a lower frequency of CKD [2].’

‘Many subjects also develop symptoms and signs of heat stress or dehydration, which is known to cause renal dysfunction. Indeed, there is some evidence that markers of acute kidney injury can occur during work shifts as well as overwork seasons [15].’

Watson slapped his knees. ‘I found a flaw. Dehydration causes a reversible type of renal disease and is not associated with structural injury. Severe heat stress can cause heat stroke in which acute tubular necrosis (ATN) occurs from hypotension or rhabdomyolysis. However, recent studies suggest the renal injury is acutely associated with fever, leukocyturia and acute interstitial nephritis. This cannot be from heat. Surely there is a toxin.’

‘Perhaps’, I replied. ‘But let me argue otherwise by discussing heat stroke, which has been recognized since Roman times [16] and for which there have been many epidemics, including in Chicago in 1995 [17], in Lyon, France in 2003 [18] and in Karachi in 2015 [19]. Heat stroke occurs when the body overheats, and the acute presentation includes fever, confusion and symptoms of dehydration. Subclinical rhabdomyolysis is common, and acute renal failure can result from leukocyturia and hematuria and nonnephrotic proteinuria, with the renal biopsy showing interstitial inflammation and tubular injury. Other features include hypokalemia, hypophosphatemia, elevated serum lactate and hyperuricemia [2021]. Subjects with heat stroke have also been reported to develop CKD over time, with biopsy showing chronic interstitial nephritis with some glomerulosclerosis [21]. These clinical signs and histologic findings are near identical to what has been reported in Mesoamerican and Sri Lankan nephropathy [2].’

‘There is also experimental evidence. Recurrent heat stress in animals induces CKD in mice that can be prevented if hydration is provided during the period of heat stress. The mechanism is mediated in part by vasopressin as well as induction of the aldose reductase–fructokinase system in the proximal tubule by hyperosmolarity. The injury involves tubular damage, local inflammation and chronic scarring, and excessive vasopressin can also induce glomerular changes as may complicate Mesoamerican nephropathy’ [22].

‘Heat stress and heat stroke are also known to be associated with hyperuricemia, acute increases in uricosuria, acidic urine and urate crystal formation [2123]. Hyperuricemia is also common in subjects with Mesoamerican nephropathy, as well as from other regions (Sri Lanka and India), and we found urate crystals in 20% of sugarcane workers at the end of the workday [1524]. On one of the hottest days of the year in 2013, we found high levels of urine uric acid and urate crystals in all seven subjects studied, which were similar to those observed in tumor lysis syndrome [25]. Thus, uricosuria may also be causing disease, either through crystalline or noncrystalline pathways [2627].’

‘This is a strong evidence, but then why is the epidemic now? This should be something that was always present,’ said Watson with a look of incredulity, as he grabbed another chocolate.

‘Well, I believe it actually has been, but that it is increasing now because of better recognition and because of climate change. While it is true that mean temperatures have only increased by 0.8–1.0°C in the last century, there has been a dramatic increase worldwide in heat extremes, which manifest as heat waves [2829]. I suspect that the heat stress–associated injury occurs most frequently during the heat waves when subjects are least prepared. In addition, rehydration with soft drinks containing fructose amplifies the injury by causing further urinary concentration and uric acid generation and providing substrate for proximal tubular fructokinase, thus providing a perfect storm [30].’

Holmes looked up at me and stared with his steely eyes. ‘And the counterarguments? One should always look for a possible alternative and provide against it. It is the first rule of criminal investigation [The Adventure of Black Peter].’

‘Of course, there are those who challenge the hypothesis. One of the biggest arguments is that it should be observed in other hot areas. As an example, some have argued that it should be in northern Sri Lanka, yet there are no reports of it there [31].’

‘Are there any reports that it is not there, specifically with screening of serum creatinines or some other careful method?’

‘No, there are no reports at all.’

‘Elementary, Johnson! Absence of evidence does not mean evidence of absence.’

‘And indeed, new hot spots are being identified every year, including in Thailand and the USA [2].’

Watson looked up. ‘But what about Cuba? I understand they have better surveillance there, and that Mesoamerican nephropathy is not observed there.’

‘Well, I do have a hypothesis.’

‘Yet another hypothesis, Johnson?’ Holmes said as he cast a mischievous look in my direction. ‘Bravo. There should be no combination of events for which the wit of man cannot conceive an explanation [The Valley of Fear].’

‘So the regulation of body temperature is largely governed by mitochondrial metabolism and by the efficiency by which mitochondria generate adenosine triphosphate (ATP). When mitochondria make ATP, they also generate heat, and these occur in opposition, in that the more effectively oxidative phosphorylation is coupled, the more ATP and the less heat that is generated, whereas if oxidative phosphorylation is “less coupled”, then more heat is generated with lesser amounts of ATP [32]. In this regard, there is evidence that Native Americans, who historically travelled across Siberia and the Bering Strait into America, acquired mutations that led to increased mitochondrial uncoupling, leading to greater heat generation during metabolism that likely provided a survival advantage in cold environments compared with Africans whose mitochondria are tightly coupled [3334]. While this would have provided a survival advantage in cold environments, in hot environments it would be expected to increase the risk for heat stress and heat stroke. Thus it could explain why Hispanics living in Central America, who have a high admixture of Native American genes, might be at increased risk for developing heat stress nephropathy while Hispanics living in Cuba, who have a higher African admixture of genes, might be relatively protected [32–35].’

‘This sounds testable’, smiled Watson.

‘Yes, one balances probabilities and chooses the most likely. It is the scientific use of the imagination [The Hound of the Baskervilles]’, Holmes said, as the smoke billowed over his head. ‘Johnson, are there any bits of evidence that confuse you?’

‘Well, yes, there are. For example, in Sri Lanka there is some evidence that areas with shallow wells have higher rates of CKD. Since shallow wells might accumulate more toxins, this argument could support the toxin argument. On the other hand, the locals there are concerned that chemicals may be in the well water, so this could lead to less water intake and greater risk for dehydration. Indeed, in the same study the intake of less water (<3 L/day), greater sun exposure and longer (>6 h/day) working hours was associated with increased risk for CKD [36]. If well water is the cause of the CKD, then lower intake would not be expected to be a risk factor.

In contrast, while most studies suggest that subjects developing Mesoamerican nephropathy tend to show evidence for dehydration [1537], there are two cross-sectional studies that report higher water intake in subjects with CKD [3839]. These data would also be consistent with a toxin in the drinking water playing a role in the epidemic. Alternatively, it could be that many of the subjects knew they had CKD and, because it is largely believed by the workers that dehydration is playing a role in the disease, they might be drinking more to protect themselves or because CKD from chronic interstitial nephritis can impair urinary concentration in subjects.’

Holmes nodded his head. ‘Circumstantial [cross-sectional] evidence is a very tricky thing,’ answered Holmes thoughtfully. ‘It may seem to point very straight to one thing, but if you shift your own point of view a little, you may find it pointing in an equally uncompromising manner to something entirely different [The Boscombe Valley Mystery].’

Watson stood up. ‘Yes, we need longitudinal data to better understand this disease. Are there not some data that low water intake in general is a risk factor for CKD?’

‘Yes, not only does low water intake predict CKD, but also a low urine pH and high serum uric acid [40–42]. High serum copeptin, which marks high vasopressin levels, also predicts the development of CKD. There are even studies that suggest bicarbonate intake may slow CKD through unknown mechanisms, which I believe might include urinary alkalinization. Thus, Mesoamerican nephropathy may simply reflect the tip of the iceberg and heat stress and dehydration may be more pervasive risk factors for CKD than we currently appreciate.’

‘Very well,’ smiled Watson, ‘you have a good argument, and most importantly, it is testable.’

Holmes nodded, ‘And remember, once you eliminate the impossible, whatever remains, no matter how improbable, must be the truth [The Sign of the Four].’

I stood up and thanked both of them and headed to the door, but turned around one last time to see Holmes had resumed reading, still blowing smoke rings in the air.

‘You know, Holmes, smoking is not good for your health.’

‘Aha, Johnson, you see but you do not observe. The distinction is clear [A Scandal in Bohemia]. I may appear to smoke, but I do not inhale. Until next time’.

I bowed my head and headed back home, where I completed a very restful sleep.

ACKNOWLEDGEMENTS

This article is considered a contribution from the Colorado Climate Consortium.

FUNDING

Dr Johnson’s research on MesoAmerican Nephropathy has been funded by the Department of Defense, the Danone Research Foundation, and the La Isla Foundation.

CONFLICT OF INTEREST STATEMENT

R.J.J. has no disclosures related to this article. He is on the scientific board of Amway and has received grants from Danone, the Department of Defense, the Veterans Administration and the National Institutes of Health. He is also a member of Colorado Research Partners, which is developing inhibitors of fructose metabolism.

(See related articles by Zoccali. Causal mechanism and component causes in Mesoamerican–Sri Lankan nephropathy: the moderator’s view. Nephrol Dial Transplant 2017; 32: 607–610; Campese. Con: Mesoamerican nephropathy: is the problem dehydration or rehydration? Nephrol Dial Transplant 2017; 32: 603–606)

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Darlene Wilkens’ branch of Spartan Insurance had flooded before, but never like this. A year ago, as she watched horrifying coverage of Hurricane Harvey from home, she realized her office in Meyerland, a low-lying part of Houston that seems to always get hit, was probably already ruined.

Wilkens, the branch manager, drove to her office a week later to find her refrigerator lying sideways in a backroom. Her file cabinets were full of “nasty” water. “It was pretty much total chaos,” she says. She lost everything except computers and some important files she’d stored high off the ground.

For three months, she worked from her house in northwest Houston where things were drier, trying to run the office remotely. She had tons of work. “There were lots of claims being called in,” she says.

As annoying and scary as Harvey was, she considers herself lucky: “It wasn’t that bad compared to people who keep losing their homes.”

In Houston, losing one’s home is becoming an all too familiar experience. Harvey dumped up to 51 inches of rain on the city during half a week, inundating more than 100,000 homes in what was the worst rainfall event in U.S. history. Then this past July 4th, 8 inches of rain fell on Houston, swamping streets, stranding unlucky motorists, and nixing the city’s Fourth of July party. By local standards, it was just a summer rainstorm.

On a Wednesday in early August, it is pouring again. Wilkens cracks open her office door and eyes the rain anxiously. “Hopefully we don’t flood,” she says.

Darlene Wilkins’ insurance business is located this strip mall in Houston’s flood-prone Meyerland neighborhood. Google EarthChances are there will be flooding — if not at her office, at some other unlucky soul’s house or business nearby. Meyerland, an upper-middle class neighborhood along Brays Bayou in inner southwest Houston, is almost entirely in the 100-year flood plain. Technically, that means it is supposed to have a 1 percent chance of flooding each year. Those numbers might sound reassuring, but recent history suggests they are not.

Houston has had at least three property-wrecking floods in the past four years. Of course, there was Harvey. The other two, in 2015 and 2016, weren’t storms severe enough to have a category assigned to them — but they also engulfed homes and businesses in low-lying Meyerland.

People here are already living the reality that communities across the country, from fire-prone neighborhoods in California to low-lying subdivisions in coastal Florida, are also facing. Not long ago, Meyerland’s 1-percent-per-year chance of flooding felt like a remote possibility. Now, as climate change ramps up storms and raises the waterline, this vulnerable neighborhood seems increasingly at risk. Mitigation projects like the ones funded by a $2.5 billion bond, expected to pass on Saturday, the one-year anniversary of Harvey’s arrival in Houston, may not be able to keep pace.

In response to the new reality, some people are leaving the neighborhood. The average home price has dropped from $395,000 in July 2017 to $347,000 a year later, according to real-estate website Zillow. Similar but less flood-prone neighborhoods haven’t seen the same drop.

But the reality is that Meyerland is still a great place to live. It’s cozy, affluent, and conveniently located, a short drive from Chinatown, Downtown, and the Museum District. So many people, like the majority of the business owners and workers in this Meyerland strip mall, are staying put.

Those holding on seem increasingly resigned to a future of incessant flooding, and they have learned to rely on their own inventions and workarounds rather than waiting for the city and Harris County to bail them out. Wilkens and her fellow businesspeople live in a dual reality: With no guarantee of a drier future, these owners and employees are trying to stay optimistic — while also preparing to flood again.

Thanks to its location on the Gulf coast and the high humidity that comes with it, Houston has always been prone to heavy rains. In most cases they have been just an annoyance.

Climate change is making the freaky outlier storms worse — and less predictable. Storms dumping 20-plus inches of rain are six times more likely than they were in the late 20th century, according to the National Academy of Sciences. On top of that, development has replaced wetlands and prairies with concrete, so stormwater goes into homes rather than sinking into the ground. According to the local nonprofit Katy Prairie Conservancy, Houston has lost 70 percent of its wetlands since 1992.

Flood maps produced by the Federal Emergency Management Agency and local agencies like the Harris County Flood Control District are supposed to help people make smart decisions about where to live or where to open a business. But they might be less useful if megastorms become more common. According to one survey along Armand Bayou, southeast of the city, the maps failed to predict 75 percent of flood claims from five recent floods.

Besides, existing business owners like the ones in the Meyerland strip mall have already put down roots. Many of them have spent years building up a local customer base.

Elaine’s Florist and Gift Baskets shortly after Hurricane Harvey (left) and today. Elaine Ousley-Nevarez / Stephen Paulsen“I’d probably retire before I move,” says Elaine Ousley-Nevarez, owner of Elaine’s Florist and Gift Baskets and a native Houstonian. Even though she had a significant loss of overall sales after Harvey, the storm actually brought in some new business. There was a surge of flower sales, as people sent condolences and thank-yous.

Ousley-Nevarez visited Europe last fall. She shows me a picture she took of a flood gauge on an old wall in a town near the border of Germany and Austria. The worst flood it had recorded was in 1501, she notes before pivoting to her point: “Flooding has been going on forever,” she says. “It is what it is. You can’t change it.”

The Harris County Flood Control District is not so fatalistic. It is trying to protect the fourth-biggest city in the United States by adding wetlands and prairies to soak up the water, and improving infrastructure like bayous and levees that can direct and control its flow. For decades, the district has been working to widen Brays Bayou in the hopes of carrying water more quickly out to the Gulf of Mexico. Lately, workers have been dredging and expanding a portion on the southern edge of Meyerland, turning an already-large artificial river into a gigantic one.

But as far as residents and business owners are concerned, Meyerland will be swamped again. It’s just a matter of when, and how bad.

“Honestly, I don’t care how wide they make [Brays Bayou] down here,” Ousley-Nevarez says. “We will still have problems.”

Reconciled to a future of floods, business owners in this little strip mall are coming up with their own ways to manage the risk. The local Game Stop, for instance, formerly relied on pallets to keep its merchandise above water during minor inundations. That’s no longer enough. Now, the store stocks sandbags in the hopes of stopping the waters.

At Los Tios restaurant a few doors down, the waters rose so fast during Harvey that employees were marooned overnight inside the popular Tex-Mex eatery. Los Tios took in more than two feet of water and closed for four months.

Chairs float in the more than two feet of water that flooded Meyerland’s Los Tios restaurant during Hurricane Harvey. Courtesy Elias DeOssio“Four months is almost fast,” says manager Elias DeOssio. “There are people who aren’t back in their homes yet.”

When it poured this year on July 4, DeOssio feared the worst, he says: “I started to really think, are we going to have a relapse?” Even if nothing disastrous happens, sales drop off as the rains keep customers at home.

Houston TranStar, the local transportation agency, announced in July it would start tracking and sharing real-time road-flooding data. The city’s streets are actually designed to flood as a last layer of infrastructure defense. The idea is that it’s better to soak cars than homes.

DeOssio already has his own system to track possible road flooding. He watches the water levels at the edge of the strip mall, where two roads meet at a low point, as an indicator of how bad it will be in the neighborhood. He also has his own damage-control plan: If it looks really stormy, he parks his 2004 Nissan Titan pickup truck on higher ground at a local school and walks half a mile in the rain to the restaurant.

“It’s wet, muddy, and you never know what critters are crawling around, trying to get out of the water,” he says, describing the conditions during a flood. Still, he says Los Tios has no plans to leave Meyerland anytime soon: “There are customers who have been with us for 50 years.”

Ousley-Nevarez, the florist, also has her own mitigation plan. She left her floors as bare concrete, so that the next time the waters rise the cleanup won’t be so difficult. She replaced her ruined wooden shelves with metal ones. She put her commercial fridge on wheels to make it easier to evacuate. As was the case with Darlene Wilkens, Ousley-Nevarez also found her fridge lying sideways in water once Harvey passed.

Ousley-Nevarez expects flood waters to come for her store again, just hopefully not at the level they did during Harvey. “We knew were going to flood,” she says of that storm. “We didn’t anticipate the amount.”

On the August Wednesday, that I visit the Meyerland stripmall, the threatening rain remains just a threat. There was brieflysome water in the streets, but for today, none in her flower shop.

Like the other businesspeople in this strip mall, Ousley-Nevarez hopes that last year’s storm was a fluke, a one-time thing. She tries not to worry unless a really big storm is coming in. But when it does — and in this part of Houston, it’s more a question of when than if — she, her metal shelves, and her concrete floor will be ready for it.

This story was originally published by Grist with the headline One year after Harvey, Houston awaits the next flood on Aug 24, 2018.

Categories: Green News
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